“Cessation of Nucleos(t)ide Analogue Therapy in HBeAg-Negative Hepatitis B Patients Increases Proinflammatory Cytokines Interferon-γ-Induced Protein 10 and Tumor Necrosis Factor during Clinical Relapse”

https://pubmed.ncbi.nlm.nih.gov/38114718

The cessation of nucleos(t)ide analogue therapy in HBeAg-negative chronic hepatitis B patients with evolving clinical relapse is associated with an increase in specific proinflammatory cytokines, interferon-γ-induced protein 10 and tumor necrosis factor.