“CHI3L1 Contributes to Coronary Artery Lesions in Kawasaki Disease-like Vasculitis Mice Model via Endothelial Cell Apoptosis Mediated by Inflammation”

https://pubmed.ncbi.nlm.nih.gov/38112139

In a mouse model of Kawasaki disease-like vasculitis, CHI3L1 may contribute to coronary artery lesions through endothelial cell apoptosis mediated by inflammatory reactions.