“CRISPR/Cas9-Mediated Nexilin Knockout in Zebrafish Reduces Cardiac Contractility and Elicits Compensation Mechanisms”

https://pubmed.ncbi.nlm.nih.gov/38114601

The CRISPR/Cas9-mediated nexilin knockout in zebrafish results in reduced cardiac contractility and some compensation mechanisms, providing a model to study nexilin’s role in heart and skeletal muscle development.