https://pubmed.ncbi.nlm.nih.gov/38091808
The MobilMS mobile education application developed for multiple sclerosis patients in Turkey improved their quality of life and was highly appreciated, accepted, and used by them.
https://pubmed.ncbi.nlm.nih.gov/38091807
The abstract reports the up-regulation of Treg-related lncRNAs, TH2-LCR, MAFTRR, NEST, RMRP, and FLICR, in Neuromyelitis Optica Spectrum Disorder (NMOSD) patients and highlights the significant diagnostic power of TH2-LCR, MAFTRR, RMRP, and FLICR lncRNAs, particularly TH2-LCR, in predicting NMOSD.
https://pubmed.ncbi.nlm.nih.gov/38091806
The abstract describes a study that identifies three distinct groups of multiple sclerosis patients based on a mixture model and predicts their trajectories over time using eight clinical and imaging parameters.
https://pubmed.ncbi.nlm.nih.gov/38091805
The Finnish Care Register for Health Care diagnoses for preeclampsia have satisfactory sensitivity and high specificity, but diagnoses for gestational diabetes and preterm delivery have lower sensitivity, limiting their use in research studies.
https://pubmed.ncbi.nlm.nih.gov/38091804
Women with stage 1 hypertension before 20 weeks’ gestation have an increased risk of progressing to stage 2 hypertension and requiring antihypertensive drug therapy within 24 hours postpartum.
https://pubmed.ncbi.nlm.nih.gov/38091802
In natural waters with humic acids and dissolved iron, 17α-ethinylestradiol can degrade due to the formation of Fe(III)-HA complexes that facilitate photodegradation through the production of reactive oxygen species, hydrogen peroxide, and Fe(II), and by maintaining higher dissolved iron concentrations and altering the Fe(III)/Fe(II) cycling.
https://pubmed.ncbi.nlm.nih.gov/38091801
The study reveals that γ-aminobutyric acid (GABA) enhances wheat’s tolerance to ozone pollution by stimulating the accumulation of flavonoids, reducing oxidative stress, and alleviating grain yield loss.
https://pubmed.ncbi.nlm.nih.gov/38091799
Environmental pollutants’ interaction with phosphoinositide signaling contributes to the onset and exacerbation of autoimmune diseases, offering potential research directions and therapeutic targets.